Evaluation and Management of Cerebral Vasospasm after Subarachnoid Hemorrhage
Hani Mohammed Alabdaly, Othman Mohammad Alassaf, Danah Kamal Kabrah, Khaled Bassam Almadi, Renda Ali Alhabib, Rakan M. Alotaibi, Abdullah Salem Almalki, Fahad Azib Alghamdi, Reema Abdullah H Albalawi, Laila Hamad Hakami, Moudhi Mohammed Alajmi
Journal of Pharmaceutical Research International · pp. 494–500 · Published 22 Dec 2021
10.9734/jpri/2021/v33i60B34645Abstract
Constriction of the large and medium-sized cerebral arteries following an aneurysmal subarachnoid haemorrhage (aSAH) is a well-known condition that primarily affects the anterior circulation supplied by the internal carotid arteries. SAH is a rare but potentially fatal type of stroke. Across the literature, authors have defined vasospasm using terms such as "symptomatic vasospasm," "delayed cerebral ischemia" (DCI), "transcranial Doppler vasospasm," and "angiographic vasospasm." Because posthemorrhagic vasospasm causes significant neurologic morbidity and death, there has been a great deal of interest and research into its physiologic basis and developing effective preventative and treatment strategies. The triple-H therapy hemodynamic augmentation technique, which includes hypertension, hemodilution, and hypervolemia, has been an important part of the treatment. In this article, we'll look at cerebral vasospasm following subarachnoid haemorrhage, including its causes, epidemiology, evaluation, and, most importantly, management.
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